PAX5 interacts with RIP2 to promote NF-κB activation and drug-resistance in B-lymphoproliferative disorders.

نویسندگان

  • Dong Wang
  • Jingyu Chen
  • Rui Li
  • Guolin Wu
  • Zimin Sun
  • Zhitao Wang
  • Zhimin Zhai
  • Fang Fang
  • Yugang Guo
  • Yongjun Zhong
  • Ming Jiang
  • Huan Xu
  • Minhua Chen
  • Guodong Shen
  • Jie Sun
  • Bailing Yan
  • Chundong Yu
  • Zhigang Tian
  • Weihua Xiao
چکیده

Paired box protein 5 (PAX5) plays a lineage determination role in B-cell development. However, high expression of PAX5 has been also found in various malignant diseases, including B-lymphoproliferative disorders (B-LPDs), but its functions and mechanisms in these diseases are still unclear. Here, we show that PAX5 induces drug resistance through association and activation of receptor-interacting serine/threonine-protein kinase 2 (RIP2; also known as RIPK2), and subsequent activation of NF-κB signaling and anti-apoptosis gene expression in B-lymphoproliferative cells. Furthermore, PAX5 is able to interact with RIP1 and RIP3, modulating both RIP1-mediated TNFR and RIP2-mediated NOD1 and NOD2 pathways. Our findings describe a new function of PAX5 in regulating RIP1 and RIP2 activation, which is at least involved in chemotherapeutic drug resistance in B-LPDs.

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عنوان ژورنال:
  • Journal of cell science

دوره 129 11  شماره 

صفحات  -

تاریخ انتشار 2016